Product references — Thiamine

Reddy, S., et al. 2010. Thiamine reduces tissue lead levels in rats: Mechanism of action. Biometals, 23 (2), 247–253. URL (abstract): http://www.ncbi.nlm.nih.gov/pubmed/20012160 (accessed 07.30.2010).

Rivas, A., et al. 2009. [Validation of questionnaires for the study of food habits and bone mass.] Nutr. Hosp., 24 (5), 521–528. URL (PDF): http://scielo.isciii.es/pdf/nh/v24n5/original1.pdf (accessed 07.30.2010).

Coelho, L., et al. 2008. Thiamin deficiency as a cause of reversible cor pulmonale. Arq. Bras. Cardiol., 91 (1), e7–e9. URL (abstract): http://www.ncbi.nlm.nih.gov/pubmed/18660937 (accessed 12.15.2008).

Lonsdale, D. 2006. A review of the biochemistry, metabolism and clinical benefits of thiamin(e) and its derivatives. Evid. Based Complement. Alternat. Med., 3 (1), 49–59. URL: http://ecam.oxfordjournals.org/cgi/content/full/3/1/49 (accessed 12.15.2008).

Haupt, E., et al. 2005. Benfotiamine in the treatment of diabetic polyneuropathy — a three-week randomized, controlled pilot study (BEDIP study). Int. J. Clin. Pharmacol. Ther., 43 (2), 71–77. URL (abstract): http://www.ncbi.nlm.nih.gov/pubmed/15726875 (accessed 12.15.2008).

McCabe–Sellers, B., et al. 2005. Diuretic medication therapy use and low thiamin intake in homebound older adults. J. Nutr. Elder., 24 (4), 57–71. URL: (abstract): http://www.ncbi.nlm.nih.gov/pubmed/16597560 (accessed 12.15.2008).

Beltramo, E., et al. 2004. Thiamine and benfotiamine prevent increased apoptosis in endothelial cells and pericytes cultured in high glucose. Diabetes Metab. Res. Rev., 20 (4), 330–336. URL: (abstract): http://www.ncbi.nlm.nih.gov/pubmed/15250036 (accessed 12.15.2008).

Shangari, N., et al. 2003. Toxicity of glyoxals — role of oxidative stress, metabolic detoxification and thiamine deficiency. Biochem. Soc. Trans., 31 (Pt. 6), 1390–1393. URL (accessed 12.15.2008).

Gibson, G., & Zhang, H. 2002. Interactions of oxidative stress with thiamine homeostasis promote neurodegeneration. Neurochem. Int., 40 (6), 493–504. URL (abstract): http://www.ncbi.nlm.nih.gov/pubmed/11850106 (accessed 12.15.2008).

Lonsdale, D., et al. 2002. Treatment of autistic spectrum children with thiamine tetrahydrofurfuryl disulfide: A pilot study. Neuroendocrinol. Lett., 23 (4), 303–308. URL (abstract): http://www.ncbi.nlm.nih.gov/pubmed/12195231 (accessed 12.15.2008).

Elmadfa, I., et al. 2001. The thiamine status of adult humans depends on carbohydrate intake. Int. J. Vitam. Nutr. Res., 71 (4), 217–221 URL: (abstract): http://www.ncbi.nlm.nih.gov/pubmed/11582856 (accessed 12.15.2008).

Lonsdale, D.2001. Sudden infant death syndrome requires genetic predisposition, some form of stress and marginal malnutrition. Med. Hypoth., 57 (3), 382–386. URL: (abstract): http://www.ncbi.nlm.nih.gov/pubmed/37452 (accessed 12.15.2008).

Schenk, G., et al. 1998. Properties and functions of the thiamin diphosphate dependent enzyme transketolase. Int. J. Biochem. Cell Biol., 30 (12), 1297–1318. URL (abstract): http://www.ncbi.nlm.nih.gov/pubmed/9924800 (accessed 12.15.2008).

Cooper, J. & Pincus, J. 1979. The role of thiamine in nervous tissue. Neurochem. Res., 4 (2), 223–239. URL: (abstract): http://www.ncbi.nlm.nih.gov/pubmed/37452 (accessed 12.15.2008).

Baker, H., et al. 1975. Inability of chronic alcoholics with liver disease to use food as a source of folates, thiamin and vitamin B6. Am. J. Clin. Nutr., 28 (12), 1377–1380. URL (PDF): http://www.ajcn.org/cgi/reprint/28/12/1377 (accessed 12.15.2008).