Product references — Vitamin B₃ (niacin, niacinamide)

Kirkland, B., et al. 2009. Niacin status and treatment-related leukemogenesis. Mol. Cancer Ther., 8 (4), 725–732. Review. URL: http://mct.aacrjournals.org/content/8/4/725.long (accessed 08.02.2010).

Tang, K., et al. 2008. Niacin deficiency causes oxidative stress in rat bone marrow cells but not through decreased NADPH or glutathione status. J. Nutr. Biochem., 19 (11), 746–753. URL (abstract): http://www.ncbi.nlm.nih.gov/pubmed/18436439 (accessed 12.16.2008).

Duan, D., et al. 2007. Protective effect of niacinamide on interleukin-1beta-induced annulus fibrosus type II collagen degeneration in vitro. J. Huazhong Univ. Sci. Technolog. Med. Sci., 27 (1), 68–71. URL (abstract): http://www.ncbi.nlm.nih.gov/pubmed/17393114 (accessed 12.16.2008).

Kostecki, L., et al. 2007. Niacin deficiency delays DNA excision repair and increases spontaneous and nitrosourea-induced chromosomal instability in rat bone marrow. Mutat. Res., 625 (1-2), 50–61. URL (abstract) http://www.ncbi.nlm.nih.gov/pubmed/17618655 (accessed 12.17.2008).

Spronck, J., et al. 2007. Niacin deficiency alters p53 expression and impairs etoposide-induced cell cycle arrest and apoptosis in rat bone marrow cells. Nutr. Cancer, 57 (1), 88–99. URL (abstract): http://www.ncbi.nlm.nih.gov/pubmed/17516866 (accessed 12.17.2008).

Fivenson, D. 2006. The mechanisms of action of nicotinamide and zinc in inflammatory skin disease. Cutis, 77 (1 Suppl.), 5–10. URL (abstract): http://www.ncbi.nlm.nih.gov/pubmed/16871773 (accessed 12.16.2008).

Niren, N. 2006. Pharmacologic doses of nicotinamide in the treatment of inflammatory skin conditions: A review. Cutis, 77 (1 Suppl.), 11–16. URL (abstract): http://www.ncbi.nlm.nih.gov/pubmed/16871774 (accessed 12.17.2008).

Kirkland, J. 2003. Niacin and carcinogenesis. Nutr. Cancer, 46 (2), 110–118. URL (abstract): http://www.ncbi.nlm.nih.gov/pubmed/14690785 (accessed 12.17.2008).

[No authors listed.] 2002. Niacinimide. Monograph. Altern. Med. Rev., 7 (6), 525–529.URL (PDF): http://www.thorne.com/altmedrev/.fulltext/7/6/525.pdf (accessed 09.01.2010).

Boyonoski, A., et al. 2002. Niacin deficiency decreases bone marrow poly(ADP-ribose) and the latency of ethylnitrosourea-induced carcinogenesis in rats. J. Nutr., 132 (1), 108–114. URL: http://jn.nutrition.org/cgi/content/full/132/1/108 (accessed 12.17.2008).

Guruprasad, K., & Vasudev, V. 2001. Inducible protective processes in animal systems: VIII. Enhancement of adaptive response by nicotinamide. Mutagenesis, 16 (3), 257–263. URL (abstract): http://www.ncbi.nlm.nih.gov/pubmed/11320152 (accessed 12.16.2008).

Hageman, G., & Stierum, R. 2001. Niacin, poly(ADP-ribose) polymerase-1 and genomic stability. Mutat. Res., 475 (1–2), 45–56. URL (abstract): http://www.ncbi.nlm.nih.gov/pubmed/11295153 (accessed 12.17.2008).

Wiencke, J. 1987. Nicotinamide deficiency in human lymphocytes prevents the [3H]thymidine-induced adaptive response for the repair of X-ray-induced chromosomal damage. Exp. Cell Res., 171 (2), 518–523. URL (abstract): http://www.ncbi.nlm.nih.gov/pubmed/2957224 (accessed 12.16.2008).

Close window